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2014

  • *Jupp L, Rivers JR, and Bhatia M. Hydrogen sulfide and substance P in acute pancreatitis. Adaptation Biology and Medicine: New Developments. (Eds. LM Popescu, AR Hargens and PK Singal). 2014; 7: 137-147.
  • *Kumar R, and Bhatia M. Cells and mediators of inflammation in acute pancreatitis. Clin Anti-Infl Anti-Allergy Drugs. 2014; 1: 11-23.
  • *Muniraj N, Ang AD, Badiei A, Rivers-Auty J, and Bhatia M. Hydrogen sulfide: a new tool to design and develop drugs. Clin Anti-Infl Anti-Allergy Drugs. 2014; 1: 57-66.
  • Badiei A, Muniraj N, Chambers S, and Bhatia M. Inhibition of hydrogen sulfide production by gene silencing attenuates inflammatory activity by down-regulation of NF-κB and MAP kinase activity in LPS-activated RAW 264.7 cells. Biomed Res Int. 2014; 2014: Article ID 848570.
  • *Sun J, and Bhatia M. Substance P at the neuro-immune crosstalk in the modulation of inflammation, asthma and antimicrobial host defense. Inflamm Allergy Drug Targets. 2014; 13: 112-120.
  • Muniraj N, Stamp LK, Badiei A, Hegde A, Cameron V, and Bhatia M. Hydrogen sulfide acts as a pro-inflammatory mediator in rheumatic disease. Int J Rheum Dis. 2014; (in press).
  • *Gaddam GR, Chambers S, and Bhatia M. ACE and ACE2 in inflammation: a tale of two enzymes. Inflamm Allergy Drug Targets. 2014; 13: 224-234.

2013

  • Badiei A, Rivers-Auty J, Ang AD, and Bhatia M. Inhibition of hydrogen sulfide production by gene silencing attenuates inflammatory activity of LPS-activated RAW 264.7 cells. Appl Microbiol Biotech. 2013; 97: 7845-7852.
  • *Rivers-Auty J, and Bhatia M. Hydrogen sulphide, systemic inflammatory response syndrome and compensatory anti-inflammatory response syndrome following sepsis. OA Inflammation 2013; 1: 2.
  • Ang AD, Rivers-Auty J, Hegde A, Ishii I, and Bhatia M. The effect of CSE gene deletion in caerulein-induced acute pancreatitis in the mouse. Am J Physiol Gastrointest Liver Physiol. 2013; 305: G712-G721.

* Invited Articles by Editor of Journal

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