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Health Sciences staff profiles

Dr Maggie Evans

PositionScientific Officer
DepartmentsDepartment of Anatomy, Department of Physiology
QualificationsBA MSc PhD
Research summaryNeuroendocrine modulation of reproductive function and energy homeostasis

Research

Investigating the neuroendocrine mechanisms that underlie the integrated control of metabolic, reproductive and circadian function, with a focus on the modulatory input of the metabolic hormones leptin and insulin.

Publications

Decourt, C., Evans, M. C., Inglis, M. A., & Anderson, G. M. (2023). Central irisin signaling is required for normal timing of puberty in female mice. Endocrinology, 164(2). doi: 10.1210/endocr/bqac208

Evans, M. C., Campbell, R. E., & Anderson, G. M. (2022). Physiological regulation of leptin as an integrative signal of reproductive readiness [Review]. Current Opinion in Pharmacology, 67, 102321. doi: 10.1016/j.coph.2022.102321

Ancel, C. M., Evans, M. C., Kerbus, R. I., Wallace, E. G., & Anderson, G. M. (2022). Deletion of PTP 1B from from brain neurons partly protects mice from diet-induced obesity and minimally improves fertility. Endocrinology, 163(2), bqab266. doi: 10.1210/endocr/bqab266

Evans, M. C., Lord, R. A., & Anderson, G. M. (2021). Multiple leptin signalling pathways in the control of metabolism and fertility: A means to different ends? International Journal of Molecular Sciences, 22, 9210. doi: 10.3390/ijms22179210

Evans, M. C., Hill, J. W., & Anderson, G. M. (2021). Role of insulin in the neuroendocrine control of reproduction [Invited]. Journal of Endocrinology, 33, e12930. doi: 10.1111/jne.12930

Evans, M. C., & Anderson, G. M. (2018). Metabolic regulation of GnRH neurons. In A. E. Herbison & T. M. Plant (Eds.), The GnRHS neuron and its control. (pp. 386-410). Hoboken, NJ: Wiley Blackwell. doi: 10.1002/9781119233275

Chapter in Book - Research

Decourt, C., Evans, M. C., Inglis, M. A., & Anderson, G. M. (2023). Central irisin signaling is required for normal timing of puberty in female mice. Endocrinology, 164(2). doi: 10.1210/endocr/bqac208

Journal - Research Article

Ancel, C. M., Evans, M. C., Kerbus, R. I., Wallace, E. G., & Anderson, G. M. (2022). Deletion of PTP 1B from from brain neurons partly protects mice from diet-induced obesity and minimally improves fertility. Endocrinology, 163(2), bqab266. doi: 10.1210/endocr/bqab266

Journal - Research Article

Evans, M. C., Campbell, R. E., & Anderson, G. M. (2022). Physiological regulation of leptin as an integrative signal of reproductive readiness [Review]. Current Opinion in Pharmacology, 67, 102321. doi: 10.1016/j.coph.2022.102321

Journal - Research Article

Mamgain, A., Sawyer, I. L., Timajo, D. A. M., Rizwan, M. Z., Evans, M. C., Ancel, C. M., Inglis, M. A., & Anderson, G. M. (2021). RFamide-related peptide neurons modulate reproductive function and stress responses. Journal of Neuroscience, 41(3), 474-488. doi: 10.1523/JNEUROSCI.1062-20.2020

Journal - Research Article

Evans, M. C., Kumar, N. S., Inglis, M. A., & Anderson, G. M. (2018). Leptin and insulin do not exert redundant control of metabolic or emotive function via dopamine neurons. Hormones & Behavior, 106, 93-104. doi: 10.1016/j.yhbeh.2018.10.001

Journal - Research Article

Evans, M. C., & Anderson, G. M. (2017). Dopamine neuron-restricted leptin receptor signaling reduces some aspects of food reward but exacerbates the obesity of leptin receptor-deficient male mice. Endocrinology, 158(12), 4246-4256. doi: 10.1210/en.2017-00513

Journal - Research Article

Evans, M. C., & Anderson, G. M. (2017). Neuroendocrine integration of nutritional signals on reproduction. Journal of Molecular Endocrinology, 58(2), R107-R128. doi: 10.1530/jme-16-0212

Journal - Research Article

Evans, M. C., Clark, V. W., Manning, P. J., De Ridder, D., & Reynolds, J. N. J. (2015). Optimizing deep brain stimulation of the nucleus accumbens in a reward preference rat model. Neuromodulation, 18(7), 531-541. doi: 10.1111/ner.12339

Journal - Research Article

Evans, M. C., Rizwan, M. Z., & Anderson, G. M. (2015). Insulin does not target CamkIIα neurones to critically regulate the neuroendocrine reproductive axis in mice. Journal of Neuroendocrinology, 27(12), 899-910. doi: 10.1111/jne.12330

Journal - Research Article

Evans, M. C., Rizwan, M. Z., & Anderson, G. M. (2014). Insulin action on GABA neurons is a critical regulator of energy balance but not fertility in mice. Endocrinology, 155(11), 4368-4379. doi: 10.1210/en.2014-1412

Journal - Research Article

Evans, M. C., Rizwan, M., Mayer, C., Boehm, U., & Anderson, G. M. (2014). Evidence that insulin signalling in gonadotrophin-releasing hormone and kisspeptin neurones does not play an essential role in metabolic regulation of fertility in mice. Journal of Neuroendocrinology, 26(7), 468-479. doi: 10.1111/jne.12166

Journal - Research Article

Rizwan, M. Z., Poling, M. C., Corr, M., Cornes, P. A., Augustine, R. A., Quennell, J. H., … Anderson, G. M. (2012). RFamide-related peptide-3 receptor gene expression in GnRH and kisspeptin neurons and GnRH-dependent mechanism of action. Endocrinology, 153(8), 3770-3779. doi: 10.1210/en.2012-1133

Journal - Research Article

Corr, M., De Souza, M. J., Toombs, R. J., & Williams, N. I. (2011). Circulating leptin concentrations do not distinguish menstrual status in exercising women. Human Reproduction, 26(3), 685-694. doi: 10.1093/humrep/deq375

Journal - Research Article

Evans, M. C., Hill, J. W., & Anderson, G. M. (2021). Role of insulin in the neuroendocrine control of reproduction [Invited]. Journal of Endocrinology, 33, e12930. doi: 10.1111/jne.12930

Journal - Research Other

Evans, M. C., Lord, R. A., & Anderson, G. M. (2021). Multiple leptin signalling pathways in the control of metabolism and fertility: A means to different ends? International Journal of Molecular Sciences, 22, 9210. doi: 10.3390/ijms22179210

Journal - Research Other

Evans, M. C., & Anderson, G. M. (2018). Integration of circadian and metabolic control of reproductive function [Mini-review]. Endocrinology, 159(11), 3661-3673. doi: 10.1210/en.2018-00691

Journal - Research Other

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