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Health Sciences staff profiles

Dr Steve Seo

PositionPostdoctoral Fellow
DepartmentDepartment of Anatomy
QualificationsPhD MSc
Research summarySynaptic transmission in neurological disorders

Research

Working with A/P Dorothy Oorschot

Publications

Seo, S., Sizemore, R. J., Reader, K. L., Smither, R. A., Wicky, H. E., Hughes, S. M., Bilkey, D. K., Parr-Brownlie, L. C., & Oorschot, D. E. (2021). A schizophrenia risk factor induces marked anatomical deficits at GABAergic-dopaminergic synapses in the rat ventral tegmental area: Essential evidence for new targeted therapies. Journal of Comparative Neurology. Advance online publication. doi: 10.1002/cne.25225

Basham, H. K., Aghoghovwia, B. E., Papaioannou, P., Seo, S., & Oorschot, D. E. (2021). Delayed double treatment with adult-sourced adipose-derived mesenchymal stem cells increases striatal medium-spiny neuronal number, decreases striatal microglial number, and has no subventricular proliferative effect, after acute neonatal hypoxia-ischemia in male rats. International Journal of Molecular Sciences, 22, 7862. doi: 10.3390/ijms22157862

Seo, S., & Leitch, B. (2017). Postnatal expression of thalamic GABAA receptor subunits in the stargazer mouse model of absence epilepsy. NeuroReport, 28(18), 1255-1260. doi: 10.1097/wnr.0000000000000909

Seo, S., & Leitch, B. (2015). Synaptic changes in GABAA receptor expression in the thalamus of the Stargazer mouse model of absence epilepsy. Neuroscience, 306, 28-38. doi: 10.1016/j.neuroscience.2015.08.021

Seo, S., & Leitch, B. (2014). Altered thalamic GABAA -receptor subunit expression in the stargazer mouse model of absence epilepsy. Epilepsia, 55(2), 224-232. doi: 10.1111/epi.12500

Basham, H. K., Aghoghovwia, B. E., Papaioannou, P., Seo, S., & Oorschot, D. E. (2021). Delayed double treatment with adult-sourced adipose-derived mesenchymal stem cells increases striatal medium-spiny neuronal number, decreases striatal microglial number, and has no subventricular proliferative effect, after acute neonatal hypoxia-ischemia in male rats. International Journal of Molecular Sciences, 22, 7862. doi: 10.3390/ijms22157862

Journal - Research Article

Seo, S., Sizemore, R. J., Reader, K. L., Smither, R. A., Wicky, H. E., Hughes, S. M., Bilkey, D. K., Parr-Brownlie, L. C., & Oorschot, D. E. (2021). A schizophrenia risk factor induces marked anatomical deficits at GABAergic-dopaminergic synapses in the rat ventral tegmental area: Essential evidence for new targeted therapies. Journal of Comparative Neurology. Advance online publication. doi: 10.1002/cne.25225

Journal - Research Article

Seo, S., & Leitch, B. (2017). Postnatal expression of thalamic GABAA receptor subunits in the stargazer mouse model of absence epilepsy. NeuroReport, 28(18), 1255-1260. doi: 10.1097/wnr.0000000000000909

Journal - Research Article

Seo, S., & Leitch, B. (2015). Synaptic changes in GABAA receptor expression in the thalamus of the Stargazer mouse model of absence epilepsy. Neuroscience, 306, 28-38. doi: 10.1016/j.neuroscience.2015.08.021

Journal - Research Article

Seo, S., & Leitch, B. (2014). Altered thalamic GABAA -receptor subunit expression in the stargazer mouse model of absence epilepsy. Epilepsia, 55(2), 224-232. doi: 10.1111/epi.12500

Journal - Research Article

Seo, S., Liu, P., & Leitch, B. (2011). Spatial learning-induced accumulation of agmatine and glutamate at hippocampal CA1 synaptic terminals. Neuroscience, 129, 28-36. doi: 10.1016/j.neuroscience.2011.07.007

Journal - Research Article

More publications...