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Bahn Lab

Lab personnel

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Research interests

My main research topic is the renal organic anion transport. Organic anions (OA) are substances, which possess a negative charge under physiological conditions. This class of substances includes a wide array of endogenous as well as exogenous compounds such as neurotransmitter metabolites, urate or prostaglandins, anti-viral drugs, antibiotics, analgesics or ochratoxin A, to name just a few. The kidney is the organ that facilitates secretion of these substances into the urine. Transport proteins involved in the secretion process of OA are members of the so called solute carrier family 22A (SLC22A). In recent years I was involved in the cloning and functional characterisation of organic anion transporters (OAT) of the SLC22A family.

Urate is the end product of purine metabolism in higher primates and well known as the substance causing gout. It has come back into clinical focus because of new studies supporting its impact on cardiovascular and neurodegenerative diseases. Recently, we have demonstrated that transporters such as organic anion transporter 4 (OAT4) or OAT10 are not only capable of urate transport, but they are also involved in renal secretion of anti-hypertensive drugs such as diuretics (furosemide or torasemide) or thiazides (hydrochlorothiazide) and even the immunosuppressant cyclosporine A. We provided evidence that OAT4 as well as OAT10 exchange these drugs during secretion against urate and are consequently involved in the hyperuricemic effect of these commonly used diuretics, thiazides and cyclosporine A (Hagos et al. 2007a; Hagos et al. 2007b; Bahn et al. 2008).

Transporters currently known to play a significant role in determining urate plasma levels are URAT1, OAT4, ABCG2 and glucose transporter 9 (GLUT9). My main current interest is the post-transcriptional regulation of these urate transporters, especially GLUT9 in general and under different stress conditions.

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Research topics

  • Organic anion transport
  • Urate transport

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Current funding

  • 2012–2013 Otago Medical Research Foundation (Laurenson grant) in collaboration with Associate Professor Lisa Stamp (Christchurch)
  • 2012–2013 The National Heart Foundation, Associate Investigator with Dr Regis Lamberts
  • 2012–2013 University of Otago Research Grant
  • 2012–2013 University of Otago Research Grant, Associate Investigator with Dr Regis Lamberts
  • 2011–2013 Prostate Cancer Foundation New Zealand
  • 2011–2012 Dean's Bequest funding
  • 2010–2013 Health Research Council (HRC) emerging researcher first grant

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Teaching

  • Kidney and Gastrointestinal Physiology

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Selected publications

Bussey, C. T., Thaung, H. P. A., Hughes, G., Bahn, A., & Lamberts, R. R. (2018). Cardiac β-adrenergic responsiveness of obese Zucker rats: The role of AMPK. Experimental Physiology. Advance online publication. doi: 10.1113/ep087054

Bahn, A. (2017). Water, electrolytes, and acid-base balance. In J. Mann & A. S. Truswell (Eds.), Essentials of human nutrition. (5th ed.) (pp. 113-130). Oxford University Press.

Purvis, N., Katare, R., & Bahn, A. (2017). The pathophysiological role of mircoRNAs in diabetic cardiac stem cells. Proceedings of the University of Otago Student Research Symposium: Te Wānaka Rakahau: Ākoka. (pp. 72). Retrieved from http://www.otago.ac.nz/graduate-research/scholarships/otago643219.html

Mugloo, S., Ashley, Z., Leader, C., Bahn, A., Sammut, I. A., Walker, R., McDonald, F. J., & Fronius, M. (2017, September). Hypertension is associated with increased ENaC expression in the arteries of Cyp1a1-Ren2 rats. Verbal presentation at the Medical Sciences Congress (MedSci), Queenstown, New Zealand.

Purvis, N. S., Bahn, A., & Katare, R. (2017, September). The pathophysiological role of microRNAs in diabetic cardiac stem cells. Verbal presentation at the Medical Sciences Congress (MedSci), Queenstown, New Zealand.

Sangkop, F., Singh, G., Rodrigues, E., Gold, E., & Bahn, A. (2016). Uric acid: A modulator of prostate cells and activin sensitivity. Molecular & Cellular Biochemistry, 414(1-2), 187-199. doi: 10.1007/s11010-016-2671-8

McDonald, F., & Bahn, A. (2016). New technologies in teaching and learning. Proceedings of the First Year Biology Educators' Colloquium (FYBC). (pp. 7). Retrieved from http://www.otago.ac.nz/fybec/programme-and-speakers/index.html

Sangkop, F., Singh, G., Rodrigues, E., Gold, E., & Bahn, A. (2016). Changes in cellular uric acid homeostasis facilitated by Glucose Transporter 9 (GLUT9) drive activin sensitivity and prostate cancer cell behaviour. Proceedings of The Physiological Society, 37, PCB129. Retrieved from http://www.physoc.org

Cooper, A., & Bahn, A. (2016). Contribution of organic anion transporters (OAT) to renal secretion of the gout medication oxypurinol. Proceedings of the Australasian Society of Clinical and Experimental Pharmacologists and Toxicologists (ASCEPT) Annual Scientific Meeting. (pp. 48). Retrieved from http://www.queenstownresearchweek.org/index.php/ascept-nz/

Shin, B., & Bahn, A. (2016, August). Is ferroptosis the driver for the onset of type-2 diabetes under hyperuricemic conditions? Poster session presented at the Medical Sciences Congress (MedSci), Nelson, New Zealand.

Cheakhun, C., Dixit, P., Katare, R., & Bahn, A. (2016, August). Role of uric acid in cardiac stem cell function. Poster session presented at the Medical Sciences Congress (MedSci), Nelson, New Zealand.

Mugloo, S., Ashley, Z., Leader, C., Bahn, A., Sammut, I., Walker, R., McDonald, F., & Fronius, M. (2016, December). Epithelial Sodium Channel (ENaC): An emerging regulator of vascular physiology. Verbal presentation at the Joint International Meeting of the Renal Scientists of the Australia and New Zealand Society of Nephrology and the Kidney in Health and Disease Network: Translational Medicine: Innovations in Renal Research, Blenheim, New Zealand.

Sangkop, F., Singh, G., Rodrigues, E., Gold, E., & Bahn, A. (2016, August-September). Changes in cellular uric acid homeostasis facilitated by glucose transporter 9 (GLUT9) drive activin sensitivity and prostate cancer cell behaviour. Verbal presentation at the Queenstown Molecular Biology (QMB) Meetings, Nelson, New Zealand.

Thaung, H. P. A., Baldi, J. C., Wang, H.-Y., Hughes, G., Cook, R. F., Bussey, C. T., Sheard, P. W., Bahn, A., Jones, P. P., Schwenke, D. O., & Lamberts, R. R. (2015). Increased efferent cardiac sympathetic nerve activity and defective intrinsic heart rate regulation in type 2 diabetes. Diabetes, 64(8), 2944-2956. doi: 10.2337/db14-0955

Thaung, H. P. A., Yao, Y., Bussey, C. T., Hughes, G., Jones, P. P., Bahn, A., Sammut, I. A., & Lamberts, R. R. (2015). Chronic bilateral renal denervation reduces cardiac hypertrophic remodelling but not β-adrenergic responsiveness in hypertensive type 1 diabetic rats. Experimental Physiology, 100(6), 628-639. doi: 10.1113/EP085021

Purvis, N., Bahn, A., & Katare, R. (2015). The role of microRNAs in cardiac stem cells. Stem Cells International, 2015(2015), 194894. doi: 10.1155/2015/194894

Knake, C., Stamp, L., & Bahn, A. (2014). Molecular mechanism of an adverse drug-drug interaction of allopurinol and furosemide in gout treatment. Biochemical & Biophysical Research Communications, 452, 157-162. doi: 10.1016/j.bbrc.2014.08.068

Lamberts, R. R., Lingam, S. J., Wang, H.-Y., Bollen, I. A. E., Hughes, G., Galvin, I. F., Bunton, R. W., Bahn, A., Katare, R., Baldi, J. C., Williams, M. J. A., Saxena, P., Coffey, S., & Jones, P. P. (2014). Impaired relaxation despite upregulated calcium-handling protein atrial myocardium from type 2 diabetic patients with preserved ejection fraction. Cardiovascular Diabetology, 13, 72. doi: 10.1186/1475-2840-13-72

Fomison-Nurse, I., Saxena, P., Coffey, S., Menon, A., Bunton, R., Galvin, I., Bahn, A., Williams, M., Cameron, V., & Katare, R. (2014). Diabetes causes the activation of the pro-ageing miRNA-34a in the human heart. Heart, Lung & Circulation, 23(Suppl. 1), (pp. e24). doi: 10.1016/j.hlc.2014.04.188

Bahn, A., Johnstone, R., & Rodrigues, E. (2014). GLUT9 facilitates the onset of type 2 diabetes mellitus. Proceedings of the Medical Sciences Congress (MedSci). (pp. 13). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Fomison-Nurse, I., Saxena, P., Menon, A., Bunton, R., Galvin, I., Bahn, A., Cameron, V., & Katare, R. (2014). Diabetes causes the activation of the pro-ageing microRNA-34a in the human heart. Proceedings of the Medical Sciences Congress (MedSci). (pp. 45). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Hall, V. A., & Bahn, A. (2014). Lithium-induced fibrosis: The role of NMDA receptors and miRNAs in renal proximal tubule cells. Proceedings of the Medical Sciences Congress (MedSci). (pp. 37). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Bahn, A., Johnstone, R., & Rodrigues, E. (2014). GLUT9 facilitates the development of type 2 diabetes mellitus under hyperuricemic conditions. Proceedings of the Physiology Society Conference. (pp. 191P). Retrieved from http://www.physiology2014.org/

Purvis, N., Bahn, A., & Katare, R. (2014). The pathophysiological role of microRNAs in diabetic cardiac stem cells. New Zealand Medical Journal, 127(1406). Retrieved from http://www.nzma.org.nz/journal

Purvis, N., Bahn, A., & Katare, R. (2014). The pathophysiological role of microRNAs in diabetic cardiac stem cells. Proceedings of the Medical Sciences Congress (MedSci). (pp. 34). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Chapter in Book - Research

Bahn, A. (2017). Water, electrolytes, and acid-base balance. In J. Mann & A. S. Truswell (Eds.), Essentials of human nutrition. (5th ed.) (pp. 113-130). Oxford University Press.

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Journal - Research Article

Bussey, C. T., Thaung, H. P. A., Hughes, G., Bahn, A., & Lamberts, R. R. (2018). Cardiac β-adrenergic responsiveness of obese Zucker rats: The role of AMPK. Experimental Physiology. Advance online publication. doi: 10.1113/ep087054

Sangkop, F., Singh, G., Rodrigues, E., Gold, E., & Bahn, A. (2016). Uric acid: A modulator of prostate cells and activin sensitivity. Molecular & Cellular Biochemistry, 414(1-2), 187-199. doi: 10.1007/s11010-016-2671-8

Thaung, H. P. A., Baldi, J. C., Wang, H.-Y., Hughes, G., Cook, R. F., Bussey, C. T., Sheard, P. W., Bahn, A., Jones, P. P., Schwenke, D. O., & Lamberts, R. R. (2015). Increased efferent cardiac sympathetic nerve activity and defective intrinsic heart rate regulation in type 2 diabetes. Diabetes, 64(8), 2944-2956. doi: 10.2337/db14-0955

Purvis, N., Bahn, A., & Katare, R. (2015). The role of microRNAs in cardiac stem cells. Stem Cells International, 2015(2015), 194894. doi: 10.1155/2015/194894

Thaung, H. P. A., Yao, Y., Bussey, C. T., Hughes, G., Jones, P. P., Bahn, A., Sammut, I. A., & Lamberts, R. R. (2015). Chronic bilateral renal denervation reduces cardiac hypertrophic remodelling but not β-adrenergic responsiveness in hypertensive type 1 diabetic rats. Experimental Physiology, 100(6), 628-639. doi: 10.1113/EP085021

Knake, C., Stamp, L., & Bahn, A. (2014). Molecular mechanism of an adverse drug-drug interaction of allopurinol and furosemide in gout treatment. Biochemical & Biophysical Research Communications, 452, 157-162. doi: 10.1016/j.bbrc.2014.08.068

Lamberts, R. R., Lingam, S. J., Wang, H.-Y., Bollen, I. A. E., Hughes, G., Galvin, I. F., Bunton, R. W., Bahn, A., Katare, R., Baldi, J. C., Williams, M. J. A., Saxena, P., Coffey, S., & Jones, P. P. (2014). Impaired relaxation despite upregulated calcium-handling protein atrial myocardium from type 2 diabetic patients with preserved ejection fraction. Cardiovascular Diabetology, 13, 72. doi: 10.1186/1475-2840-13-72

Dinour, D., Bahn, A., Ganon, L., Ron, R., Geifman-Holtzman, O., Knecht, A., … Holtzman, E. J. (2011). URAT1 mutations cause renal hypouricemia type 1 in Iraqi Jews. Nephrology Dialysis Transplantation, 26(7), 2175-2181. doi: 10.1093/ndt/gfq722

Rödiger, M., Zhang, X., Ugele, B., Gersdorff, N., Wright, S. H., Burckhardt, G., & Bahn, A. (2010). Organic anion transporter 3 (OAT3) and renal transport of the metal chelator 2,3-dimercapto-1-propanesulfonic acid (DMPS). Canadian Journal of Physiology & Pharmacology, 88(2), 141-146. doi: 10.1139/Y09-123

Bakhiya, N., Arlt, V. M., Bahn, A., Burckhardt, G., Phillips, D. H., & Glatt, H. (2009). Molecular evidence for an involvement of organic anion transporters (OATs) in aristolochic acid nephropathy. Toxicology, 264(1-2), 74-79. doi: 10.1016/j.tox.2009.07.014

Bahn, A., Hagos, Y., Reuter, S., Balen, D., Brzica, H., Krick, W., … Burckhardt, G. (2008). Identification of a new urate and high affinity nicotinate transporter: hOAT10 (SCL22A13). Journal of Biological Chemistry, 283(24), 16332-16341. doi: 10.1074/jbc.M800737200

Ugele, B., Bahn, A., & Rex-Haffner, M. (2008). Functional differences in steroid sulfate uptake of organic anion transporter 4 (OAT4) and organic anion transporting polypeptide 2B1 (OATP2B1) in human placenta. Journal of Steroid Biochemistry & Molecular Biology, 111(1-2), 1-6. doi: 10.1016/j.jsbmb.2008.04.001

Sabolić, I., Asif, A. R., Budach, W. E., Wanke, C., Bahn, A., & Burckhardt, G. (2007). Gender differences in kidney function. Pflügers Archiv: European Journal of Physiology, 455(3), 397-429. doi: 10.1007/s00424-007-0308-1

Hagos, Y., Stein, D., Ugele, B., Burckhardt, G., & Bahn, A. (2007). Human renal organic anion transporter 4 operates as an asymmetric urate transporter. Journal of the American Society of Nephrology, 18(2), 430-439. doi: 10.1681/ASN.2006040415

Ljubojević, M., Balen, D., Breljak, D., Kušan, M., Anzai, N., Bahn, A., … Sabolić, I. (2007). Renal expression of organic anion transporter OAT2 in rats and mice is regulated by sex hormones. American Journal of Physiology: Renal Physiology, 292(1), F361-F372. doi: 10.1152/ajprenal.00207.2006

Hagos, Y., Bahn, A., Vormfelde, S. V., Brockmöller, J., & Burckhardt, G. (2007). Torasemide transport by organic anion transporters contributes to hyperuricemia. Journal of the American Society of Nephrology, 18(12), 3101-3109. doi: 10.1681/ASN.2007010106

Bakhiya, N., Stephani, M., Bahn, A., Ugele, B., Seidel, A., Burckhardt, G., & Glatt, H. (2006). Uptake of chemically reactive, DNA-damaging sulfuric acid esters into renal cells by human organic anion transporters. Journal of the American Society of Nephrology, 17(5), 1414-1421. doi: 10.1681/ASN.2005080801

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Journal - Research Other

Ljubojević, M., Balen, D., Breljak, D., Kusan, M., Anzai, N., Bahn, A., … Sabolic, I. (2007). Corrigendum to "Renal expression of organic anion transporter OAT2 in rats and mice is regulated by sex hormones" [Am. J. Physiol. Renal Physiol. 292(1): F361-F372]. American Journal of Physiology: Renal Physiology, 292(4), F1302. doi: 10.1152/ajprenal.zh2-4716-corr.2007

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Conference Contribution - Published proceedings: Abstract

Purvis, N., Katare, R., & Bahn, A. (2017). The pathophysiological role of mircoRNAs in diabetic cardiac stem cells. Proceedings of the University of Otago Student Research Symposium: Te Wānaka Rakahau: Ākoka. (pp. 72). Retrieved from http://www.otago.ac.nz/graduate-research/scholarships/otago643219.html

McDonald, F., & Bahn, A. (2016). New technologies in teaching and learning. Proceedings of the First Year Biology Educators' Colloquium (FYBC). (pp. 7). Retrieved from http://www.otago.ac.nz/fybec/programme-and-speakers/index.html

Cooper, A., & Bahn, A. (2016). Contribution of organic anion transporters (OAT) to renal secretion of the gout medication oxypurinol. Proceedings of the Australasian Society of Clinical and Experimental Pharmacologists and Toxicologists (ASCEPT) Annual Scientific Meeting. (pp. 48). Retrieved from http://www.queenstownresearchweek.org/index.php/ascept-nz/

Sangkop, F., Singh, G., Rodrigues, E., Gold, E., & Bahn, A. (2016). Changes in cellular uric acid homeostasis facilitated by Glucose Transporter 9 (GLUT9) drive activin sensitivity and prostate cancer cell behaviour. Proceedings of The Physiological Society, 37, PCB129. Retrieved from http://www.physoc.org

Fomison-Nurse, I., Saxena, P., Coffey, S., Menon, A., Bunton, R., Galvin, I., Bahn, A., Williams, M., Cameron, V., & Katare, R. (2014). Diabetes causes the activation of the pro-ageing miRNA-34a in the human heart. Heart, Lung & Circulation, 23(Suppl. 1), (pp. e24). doi: 10.1016/j.hlc.2014.04.188

Purvis, N., Bahn, A., & Katare, R. (2014). The pathophysiological role of microRNAs in diabetic cardiac stem cells. Proceedings of the Medical Sciences Congress (MedSci). (pp. 34). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Purvis, N., Bahn, A., & Katare, R. (2014). The pathophysiological role of microRNAs in diabetic cardiac stem cells. New Zealand Medical Journal, 127(1406). Retrieved from http://www.nzma.org.nz/journal

Bahn, A., Johnstone, R., & Rodrigues, E. (2014). GLUT9 facilitates the development of type 2 diabetes mellitus under hyperuricemic conditions. Proceedings of the Physiology Society Conference. (pp. 191P). Retrieved from http://www.physiology2014.org/

Hall, V. A., & Bahn, A. (2014). Lithium-induced fibrosis: The role of NMDA receptors and miRNAs in renal proximal tubule cells. Proceedings of the Medical Sciences Congress (MedSci). (pp. 37). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Fomison-Nurse, I., Saxena, P., Menon, A., Bunton, R., Galvin, I., Bahn, A., Cameron, V., & Katare, R. (2014). Diabetes causes the activation of the pro-ageing microRNA-34a in the human heart. Proceedings of the Medical Sciences Congress (MedSci). (pp. 45). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Bahn, A., Johnstone, R., & Rodrigues, E. (2014). GLUT9 facilitates the onset of type 2 diabetes mellitus. Proceedings of the Medical Sciences Congress (MedSci). (pp. 13). Retrieved from http://www.physoc.org.nz/meetings/archive/medsci-2014-abstracts

Kalita, P., Bahn, A., Bedford, J., Leader, J., & Walker, R. (2013). Long-term modulation of lithium-induced nephrogenic diabetes isipidus by amiloride. Nephrology, 18(Suppl. 1), (pp. 37). doi: 10.1111/nep.12121

Stamp, L. K., Knake, C., & Bahn, A. (2013). A clinically significant interaction between furosemide and allopurinol: Potential implications for clinical practice. Arthritis & Rheumatism, 65(10, Suppl.), (pp. S496). doi: 10.1002/art.38216

Kalita, P., Bahn, A., Bedford, J., Leader, J., & Walker, R. (2013). Amiloride modifies renal fibrosis induced by long term lithium treatment. Nephrology, 18(Suppl. 1), (pp. 57). doi: 10.1111/nep.12121

Rodrigues, E., & Bahn, A. (2013). Impact of urate on prostate cancer development. Proceedings of the International Union of Physiological Sciences (IUPS) Congress. (pp. 547P). Retrieved from http://edition.pagesuite-professional.co.uk/launch.aspx?pnum=798&EID=3a1c456e-d4f7-4725-98f6-b1d998184a5e

Thaung, H. P. A., Yao, Y., Bahn, A., Sammut, I. A., & Lamberts, R. R. (2012). Renal denervation does not restore the attenuated cardiac response to β-adrenergic stimulation in the hypertensive diabetic rat. Heart, Lung & Circulation, 21(12), (pp. 859-860). doi: 10.1016/j.hlc.2012.08.022

Hagos, Y., Burckhardt, G., & Bahn, A. (2007). Identification of a human organic anion transporter (hORCTL3, SLC22A13) facilitating urate and high affinity nicotinate exchange in kidney and intestine. FASEB Journal, 21, (pp. A910). [Abstract]

Hagos, Y., Bahn, A., Vormfelde, S. V., Brockmoller, J., & Burckhardt, G. (2007). Interaction of human organic anion transporters with the loop diuretic torasemide and the impact on renal urate excretion. FASEB Journal, 21, (pp. A909-A910). [Abstract]

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Conference Contribution - Poster Presentation (not in published proceedings)

Shin, B., & Bahn, A. (2016, August). Is ferroptosis the driver for the onset of type-2 diabetes under hyperuricemic conditions? Poster session presented at the Medical Sciences Congress (MedSci), Nelson, New Zealand.

Cheakhun, C., Dixit, P., Katare, R., & Bahn, A. (2016, August). Role of uric acid in cardiac stem cell function. Poster session presented at the Medical Sciences Congress (MedSci), Nelson, New Zealand.

Kalita, P., Bahn, A., Bedford, J., Leader, J., & Walker, R. (2013, September). Long term amiloride therapy attenuates lithium-induced kidney interstitial fibrosis. Poster session presented at the Division of Health Sciences Research Forum: Foundations for Health: Otago Health Research, Christchurch, New Zealand.

Johnstone, R., Rodrigues, E., & Bahn, A. (2013, September). Impact of urate on pancreatic beta cell function and the development of diabetes mellitus. Poster session presented at the Division of Health Sciences Research Forum: Foundations for Health: Otago Health Research, Christchurch, New Zealand.

Fomison-Nurse, I., Saxena, P., Menon, A., Bunton, R., Galvin, I., Bahn, A., & Katare, R. (2013, August). Diabetes causes the activation of the pro-ageing miRNA-34a in the human heart. Poster session presented at the Medical Sciences Congress (MedSci), Queenstown, New Zealand.

Knake, C., Stamp, L., & Bahn, A. (2012, August). Furosemide and alloprinol: An adverse drug interaction with clinical implications for treatment of gout in hypertensive patients. Poster session presented at the Translational Nephrology Conference: From Mechanics to Therapeutics, Queenstown, New Zealand.

Knake, C., Stamp, L., & Bahn, A. (2012, September). Furosemide and allopurinol: An adverse drug interaction with clinical implications for treatment of gout in hypertensive patients. Poster session presented at the Division of Health Sciences Research Forum: Health Matters: Research Excellence at Otago, Dunedin, New Zealand.

Rodrigues, E., & Bahn, A. (2012, August). Expression of ZIP1 and ZIP3 in prostate cancer cells and their regulation via miRNAs. Poster session presented at the Queenstown Molecular Biology (QMB) Meetings, Queenstown, New Zealand.

Rodrigues, E., & Bahn, A. (2012, August). miRNA-153 is a regulator of GLUT9 expression in a urate dependent manner. Poster session presented at the Queenstown Molecular Biology (QMB) Meetings, Queenstown, New Zealand.

Johnstone, R., Rodrigues, E., & Bahn, A. (2012, August). Interaction of GLUT9 with GLUT2 and its overall role in glucose homeostasis. Poster session presented at the Queenstown Molecular Biology (QMB) Meetings, Queenstown, New Zealand.

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Conference Contribution - Verbal presentation and other Conference outputs

Mugloo, S., Ashley, Z., Leader, C., Bahn, A., Sammut, I. A., Walker, R., McDonald, F. J., & Fronius, M. (2017, September). Hypertension is associated with increased ENaC expression in the arteries of Cyp1a1-Ren2 rats. Verbal presentation at the Medical Sciences Congress (MedSci), Queenstown, New Zealand.

Purvis, N. S., Bahn, A., & Katare, R. (2017, September). The pathophysiological role of microRNAs in diabetic cardiac stem cells. Verbal presentation at the Medical Sciences Congress (MedSci), Queenstown, New Zealand.

Mugloo, S., Ashley, Z., Leader, C., Bahn, A., Sammut, I., Walker, R., McDonald, F., & Fronius, M. (2016, December). Epithelial Sodium Channel (ENaC): An emerging regulator of vascular physiology. Verbal presentation at the Joint International Meeting of the Renal Scientists of the Australia and New Zealand Society of Nephrology and the Kidney in Health and Disease Network: Translational Medicine: Innovations in Renal Research, Blenheim, New Zealand.

Sangkop, F., Singh, G., Rodrigues, E., Gold, E., & Bahn, A. (2016, August-September). Changes in cellular uric acid homeostasis facilitated by glucose transporter 9 (GLUT9) drive activin sensitivity and prostate cancer cell behaviour. Verbal presentation at the Queenstown Molecular Biology (QMB) Meetings, Nelson, New Zealand.

Fomison-Nurse, I., Saxena, P., Menon, A., Bunton, R., Galvin, I., Bahn, A., & Katare, R. (2013, August). Diabetes causes the activation of the pro-ageing miRNA-34a in the human heart. Verbal presentation at the Queenstown Molecular Biology (QMB) Meetings, Queenstown, New Zealand.

Kalita, P., Bahn, A., Walker, R., Leader, J., & Bedford, J. (2012, August). Mechanism underlying kidney fibrosis in rats due to prolonged lithium treatment. Verbal presentation at the Translational Nephrology Conference: From Mechanics to Therapeutics, Queenstown, New Zealand.

Bahn, A. (2011, February). miRNAs powerful regulators of protein expression as molecular targets for drugs. Invited presentation at the New Zealand Chapter of the Controlled Release Society 13th Conference on Formulation and Delivery of Bioactives, Dunedin, New Zealand.

Bahn, A. (2010, August-September). Comparison of urate transport via OAT2 in different species. Verbal presentation at the Queenstown Molecular Biology (QMB) Meetings, Queenstown, New Zealand.

Bahn, A. (2010, November-December). OAT2 is a urate transporter in different species. Verbal presentation at the Medical Sciences Congress: A New Decade of Discovery (MedSciNZ), Queenstown, New Zealand.

More publications...

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