Red X iconGreen tick iconYellow tick icon

Principal Investigator

Greg Anderson 186x228 Professor Greg Anderson

Professor Greg Anderson's biography
Tel +64 3 479 7320
Fax +64 3 479 7254
Department of Anatomy website

Research interests

My research group's interests are focused on the regulation of fertility by hormones such as leptin, insulin that provide the brain with information about the body's nutritional status, and on the effects of a recently discovered group of brain cells called RFRP neurons on fertility and stress responses (such as anxiety). Beyond our focused basic research projects, our 'big picture' goal is the development of new and improved treatments for infertility and anxiety-related disorders; both of which are unseen conditions experienced by a large proportion of modern societies. For example, we are currently developing drugs that combat the anxiety-promoting effects of RFRP neurons. The Anderson Research Group is based in the Department of Anatomy and forms one of the laboratories that make up University of Otago's Centre for Neuroendocrinology.

Clinical conditions

  • Infertility
  • Metabolic syndrome
  • Chronic anxiety disorders

Professor Anderson's researcher profile

List of publications for Greg Anderson (Google Scholar)

Anderson Laboratory staff and students

Caroline Decourt_webDr Caroline Decourt, Postdoctoral Fellow
Neuroendocrine requirements for puberty onset

Megan InglisMegan Inglis, Research Technician

Rebecca Lord_webRebecca Lord, PhD student
Signalling pathways used by the hormone leptin to support puberty

Sushanth Yadhav, Masters student

Kennedy Stevenson, PgDipSci student

Zsaleya Sword-Tua, Honours student

Visit Our people for an alphabetical list of all Centre for Neuroendocrinology staff.

Research Funders


Evans, M. C., & Anderson, G. M. (2023). The role of RFRP neurons in the allostatic control of reproductive function. International Journal of Molecular Sciences, 24(21), 15851. doi: 10.3390/ijms242115851

Lord, R. A., Inglis, M. A., & Anderson, G. M. (2023). Neuronal deletion of STAT3, but not ERK2, causes obesity and delayed puberty onset in mice. In T. Milne, M. Anwar, L. Burga, H. Harcombe, M. Garelja, A. Middleton, D. C. Ribeiro, N. Fleming, K. Ogbuehi & A. Bahn (Eds.), Proceedings of the 267th Otago Medical School Research Society (OMSRS) Meeting: PhD Student Speaker Awards. Dunedin, New Zealand: OMSRS. Retrieved from

Kerbus, R. I., Inglis, M. A., & Anderson, G. M. (2023). Neuronal Ptpn1 and Socs3 deletion improves metabolism but not anovulation in a mouse PCOS model. Journal of Endocrinology. Advance online publication. doi: 10.1530/JOE-23-0023

Evans, M. C., Wallace, E. G., Ancel, C. M., & Anderson, G. M. (2023). Leptin, but not estradiol, signaling in PACAP neurons modulates puberty onset. Endocrinology, 164(8), bqad097. doi: 10.1210/endocr/bqad097

Aquino, N. S. S., Mansano, N. S., Vieira, F. A. S., Silva, K. S. C., Gusmao, D. O., Anderson, G. M., … Szawka, R. E. (2023). RFamide-related peptide 3 signaling via neuropeptide FF receptor stimulates prolactin secretion in female rats. Endocrinology. Advance online publication. doi: 10.1210/endocr/bqad102

Back to top