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Contact Details

Email
kyoko.potapov@otago.ac.nz
Position
Assistant Research Fellow
Department
Department of Physiology

Research

Research to investigate the central neuroendocrine abnormalities of polycystic ovarian syndrome (PCOS), the leading cause of infertility in women.

Publications

McLoughlin, J., Sayfullaeva, J., Kiliç, E., Carmody, C., Grochowska, N., Potapov, K., Potapov, D., Clarkson, A., Peppercorn, K., Tate, W., & Kwakowsky, A. (2026). Estren prevents beta-amyloid-induced basal forebrain cholinergic loss and long-term spatial memory deficits in aged female mice. Scientific Reports. Advance online publication. doi: 10.1038/s41598-026-49638-1 Journal - Research Article

Glendining, K. A., Prescott, M., Potapov, K., Kip, E., & Campbell, R. E. (2026). Arcuate nucleus-specific progesterone receptor knockdown in female mice is sufficient to induce PCOS-like hyperactivity in the reproductive axis. Journal of Neuroendocrinology, 38(4), e70181. doi: 10.1111/jne.70181 Journal - Research Article

Lott, E. E. A., Prescott, M., Potapov, K., Handelsman, D. J., Glendining, K. A., & Campbell, R. E. (2025). Forebrain AR deletion restores PR expression but not reproduction in prenatally androgenised female mice. Endocrinology. Advance online publication. doi: 10.1210/endocr/bqaf161 Journal - Research Article

Lott, E., Prescott, M., Potapov, K., Glendining, K., & Campbell, R. E. (2024, August-September). Androgen receptor expression in neurons is not required to program reproductive dysfunction in polycystic ovary syndrome-like mice. Verbal presentation at the 18th New Zealand Medical Sciences Congress (MedSci), Queenstown, New Zealand. Conference Contribution - Verbal presentation and other Conference outputs

Hames, M., Potapov, K., Ohline, S. M., & Jones, P. P. (2024). Investigating the effect of CK2 phosphorylation of RyR2 on neuronal excitability. In K.-L. Horne (Ed.), Proceedings of the 40th International Australasian Winter Conference on Brain Research (AWCBR). (pp. 49). Retrieved from https://www.awcbr.org Conference Contribution - Published proceedings: Abstract

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